## Anatomical Basis of the Deficit The patient presents with **contiguous motor weakness** affecting the face, arm, and leg on the right side (contralateral to the left-sided lesion). This pattern is pathognomonic for a lesion affecting the **corticospinal tract**, which passes through the posterior limb of the internal capsule. ### Internal Capsule Organization | Limb | Contents | Clinical Deficit if Damaged | |------|----------|-----------------------------| | **Anterior** | Thalamocortical radiations (frontal lobe), frontopontine fibres | Personality changes, cognitive deficits, dysarthria | | **Genu** | Corticobulbar fibres (CN motor nuclei) | Facial weakness, dysarthria, dysphagia | | **Posterior** | Corticospinal tract, sensory radiations | Contralateral hemiparesis (face, arm, leg), hemisensory loss | | **Retrolenticular** | Optic radiations | Contralateral homonymous hemianopia | ### Why Posterior Limb? **Key Point:** The corticospinal tract (pyramidal tract) descends from the motor cortex → internal capsule (posterior limb) → cerebral peduncle → pons → medullary pyramids → decussation → spinal cord. A single lesion in the posterior limb produces **contiguous motor loss** of the entire contralateral body. **Clinical Pearl:** The "pure motor stroke" pattern (face + arm + leg weakness without sensory loss or visual field defect) is the classic presentation of posterior limb internal capsule infarction, typically from lacunar stroke in hypertensive patients. **High-Yield:** The posterior limb is the most commonly affected region in lacunar stroke due to its vascular territory (lenticulostriate arteries from the middle cerebral artery). [cite:Snell's Neuroanatomy 8e Ch 10] 
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