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    Subjects/Dermatology/Stevens-Johnson Syndrome and TEN
    Stevens-Johnson Syndrome and TEN
    hard
    hand Dermatology

    A 35-year-old man with HIV infection (CD4 count 180 cells/μL) presents with a 3-day history of fever, sore throat, and a widespread erythematous maculopapular rash involving his trunk and extremities. He was started on trimethoprim-sulfamethoxazole (TMP-SMX) prophylaxis 10 days ago for Pneumocystis jirovecii pneumonia (PCP) prevention. On examination, he has painful oral ulcers, conjunctival injection, and scattered target lesions. Skin biopsy shows full-thickness epidermal necrosis with subepidermal blister formation and scattered apoptotic keratinocytes. What is the most likely diagnosis and the most appropriate next step?

    A. Drug reaction with eosinophilia and systemic symptoms (DRESS); continue TMP-SMX and add systemic corticosteroids
    B. Acute HIV exanthem; switch to alternative PCP prophylaxis and monitor clinically
    C. Stevens-Johnson Syndrome (SJS); discontinue TMP-SMX immediately and initiate ICU-level supportive care with consideration of IVIG
    D. Erythema multiforme major; use topical corticosteroids and continue TMP-SMX with antihistamines

    Explanation

    ## Diagnosis: Stevens-Johnson Syndrome (SJS) ### Histopathological Confirmation **Key Point:** The biopsy findings are diagnostic: - **Full-thickness epidermal necrosis** — hallmark of SJS/TEN - **Subepidermal blister formation** — due to epidermal-dermal separation - **Scattered apoptotic keratinocytes** — reflects TNF-α and Fas-mediated apoptosis - **Minimal dermal inflammation** — distinguishes SJS/TEN from erythema multiforme (which shows dense dermal infiltrate) This patient has **SJS** (not TEN) because the rash involves <10% BSA, but the pathology is identical to TEN—just less extensive. ### Clinical Context: Why TMP-SMX in an HIV Patient? **High-Yield:** Patients with advanced HIV (CD4 <200 cells/μL) are at **markedly increased risk** of SJS/TEN from TMP-SMX: - Impaired immune tolerance and altered drug metabolism - Slower acetylation of drug metabolites - Increased formation of reactive metabolites (arylamine) - TMP-SMX is the **most common cause of SJS/TEN in HIV-positive patients** ### Differential Diagnosis Table | Feature | SJS | DRESS | Acute HIV Exanthem | EM Major | | --- | --- | --- | --- | --- | | **Onset** | 1–3 weeks post-drug | 2–8 weeks post-drug | Days to weeks | Variable | | **Mucosal involvement** | Yes (>90%) | Rare | Rare | Yes | | **Target lesions** | True targets (3 zones) | Absent | Absent | True targets | | **Histology** | Full-thickness epidermal necrosis | Perivascular infiltrate, eosinophils | Superficial perivascular infiltrate | Dermal infiltrate, minimal epidermal necrosis | | **Systemic symptoms** | Fever, malaise | Fever, lymphadenopathy, hepatosplenomegaly | Fever, lymphadenopathy | Usually absent | | **Eosinophilia** | Absent or mild | **Present (>1,500/μL)** | Absent | Absent | | **Liver involvement** | Rare | Common (hepatitis) | Rare | Absent | | **Management** | Drug withdrawal, supportive care ± IVIG | Drug withdrawal, systemic corticosteroids | Supportive care | Topical/systemic corticosteroids | ### Management Algorithm for SJS ```mermaid flowchart TD A[SJS confirmed by biopsy]:::outcome --> B[Discontinue offending drug<br/>TMP-SMX]:::urgent B --> C{BSA involved<br/>and severity?}:::decision C -->|<10% BSA, stable vitals| D[High-dependency unit<br/>or ICU admission]:::action C -->|≥10% BSA| E[ICU admission<br/>Burn unit consultation]:::action D --> F[Fluid/electrolyte management<br/>Nutritional support<br/>Infection prevention]:::action E --> F F --> G{Consider adjunctive<br/>therapy?}:::decision G -->|Early presentation| H[IVIG 2 g/kg over 3-5 days<br/>OR Systemic corticosteroids]:::action G -->|Late presentation| I[Supportive care alone<br/>Monitor for complications]:::action H --> J[Ophthalmology consult<br/>Monitor for blindness]:::action I --> J ``` ### Immediate Management Steps 1. **Discontinue TMP-SMX immediately** — no exceptions 2. **Admit to ICU or high-dependency unit** — even with <10% BSA, SJS requires close monitoring 3. **Fluid resuscitation** — use Parkland formula: 4 mL × %BSA × weight (kg) over first 24 hours 4. **Ophthalmology consultation** — conjunctival involvement can lead to symblepharon and blindness if not managed urgently 5. **Nutritional support** — enteral feeding preferred; avoid unnecessary antibiotics 6. **Consider IVIG** — 2 g/kg IV over 3–5 days if within 48 hours of rash onset; may halt disease progression 7. **Alternative PCP prophylaxis** — once stable, switch to dapsone or atovaquone-proguanil (avoid sulfonamides permanently) ### Why NOT DRESS? **Warning:** DRESS (Drug Reaction with Eosinophilia and Systemic Symptoms) is a common distractor: - DRESS typically presents 2–8 weeks after drug initiation; this patient developed rash at 10 days (earlier) - DRESS features hepatosplenomegaly, lymphadenopathy, and **eosinophilia >1,500/μL** — not mentioned here - DRESS histology shows perivascular infiltrate with eosinophils, NOT full-thickness epidermal necrosis - DRESS is managed with drug withdrawal + systemic corticosteroids; SJS requires drug withdrawal + supportive care ± IVIG ### Why NOT Acute HIV Exanthem? **Clinical Pearl:** Acute HIV exanthem (part of acute retroviral syndrome) is a maculopapular rash without mucosal involvement, target lesions, or systemic toxicity. Histology shows superficial perivascular infiltrate, not epidermal necrosis. This patient's presentation is far too severe. ### Why NOT Erythema Multiforme Major? **Key Point:** EM major and SJS are **distinct entities** despite superficial overlap: - **EM major:** Typically triggered by HSV recurrence (not drugs); lesions are true targets with 3 zones; histology shows **dermal infiltrate with minimal epidermal necrosis**; mucosal involvement is present but less severe - **SJS:** Drug-triggered; full-thickness epidermal necrosis on biopsy; severe mucosal involvement; systemic toxicity - **Management differs:** EM major responds to topical corticosteroids and HSV prophylaxis; SJS requires drug withdrawal and ICU-level care This biopsy showing **full-thickness epidermal necrosis** rules out EM major. ### Special Consideration: HIV + TMP-SMX **High-Yield:** TMP-SMX is the **most common cause of SJS/TEN in HIV patients**, especially with CD4 <200 cells/μL. The risk is 100–1,000 times higher than in immunocompetent individuals. Once SJS/TEN develops from TMP-SMX, the patient must **never receive sulfonamides again**. Alternative PCP prophylaxis options: - **Dapsone** (if no sulfonamide allergy) - **Atovaquone-proguanil** - **Pentamidine** (inhaled or IV) - **Clindamycin-primaquine** ![Stevens-Johnson Syndrome and TEN diagram](https://mmcphlazjonnzmdysowq.supabase.co/storage/v1/object/public/blog-images/explanation/24880.webp)

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