## Distinguishing Placental Abruption from Placenta Previa ### Key Clinical Discriminator **Key Point:** Uterine tenderness and evidence of maternal hypovolemic shock are hallmark features of placental abruption, distinguishing it from placenta previa. ### Comparative Table | Feature | Placental Abruption | Placenta Previa | |---------|-------------------|------------------| | **Uterine tenderness** | Present (prominent) | Absent | | **Maternal shock** | Common (concealed bleeding) | Rare (visible bleeding) | | **Vaginal bleeding** | May be concealed (retroplacental) | Always visible | | **Fetal distress** | Common (due to placental insufficiency) | Variable | | **Uterine contractions** | Present, often tetanic | Absent (relaxed uterus) | | **Coagulopathy** | Frequent (DIC) | Absent | ### Clinical Pearl **Clinical Pearl:** In placental abruption, the bleeding is often *concealed* behind the placenta, leading to a discrepancy between visible vaginal bleeding and degree of maternal shock. Uterine tenderness reflects the retroplacental hematoma and irritation of the myometrium. In contrast, placenta previa presents with *visible* vaginal bleeding without uterine tenderness or shock (unless massive hemorrhage occurs). ### High-Yield Distinction **High-Yield:** The **tetanic uterus + tenderness + shock** triad is pathognomonic for abruption and mandates emergency cesarean delivery. Placenta previa typically presents with painless vaginal bleeding and a soft, relaxed uterus. ### Pathophysiology In abruption, the placenta separates prematurely from the decidua, causing: 1. Retroplacental hematoma formation → uterine irritation and tenderness 2. Concealed hemorrhage → maternal hypovolemia and shock disproportionate to visible bleeding 3. Release of thromboplastin → consumptive coagulopathy (DIC) 4. Placental insufficiency → fetal hypoxia and distress In previa, the placenta is abnormally implanted over the cervical os but remains attached; bleeding is from exposed placental vessels at the cervix and is visible. 
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